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How does the nervous system carry rapid signals around the body?

Describe the structure of neurons and explain how nerve impulses and synaptic transmission carry information

Neurons carry electrical impulses (action potentials) along axons and pass signals across synapses using neurotransmitters, providing fast coordination in homeostasis.

Generated by Claude Opus 4.79 min answer

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  1. What this dot point is asking
  2. Neuron structure
  3. The nerve impulse (action potential)
  4. The synapse

What this dot point is asking

You need to describe neuron structure, explain how a nerve impulse (action potential) is generated and travels, and explain how signals cross synapses. This is the fast communication pathway used in homeostasis.

Neuron structure

A neuron (nerve cell) is specialised to carry electrical signals. Its main parts are:

  • Dendrites - receive signals and carry them toward the cell body.
  • Cell body - contains the nucleus.
  • Axon - a long fibre that carries the impulse away from the cell body.
  • Myelin sheath - a fatty insulating layer around many axons that speeds up impulse transmission.
  • Axon terminals - pass the signal on at synapses.

The three functional types are sensory neurons (receptor to central nervous system), motor neurons (central nervous system to effector), and interneurons (relay neurons) that connect them.

The nerve impulse (action potential)

A nerve impulse is an electrical signal called an action potential.

  • Resting potential. A resting neuron is polarised: the inside is negative relative to the outside, maintained by the sodium-potassium pump (active transport) moving ions across the membrane.
  • Depolarisation. When a stimulus is strong enough to reach the threshold, sodium ions rush into the neuron, reversing the charge. This is the action potential.
  • Propagation. The depolarisation triggers the next section of membrane, so the impulse travels along the axon as a wave.
  • Repolarisation. Potassium ions move out and the pumps restore the resting potential, ready for the next impulse.

The action potential is all-or-nothing: if the threshold is reached the impulse fires fully; if not, nothing happens. A stronger stimulus produces more frequent impulses, not bigger ones. Myelinated axons conduct faster because the impulse jumps between gaps in the sheath.

The synapse

Neurons are not physically joined; there is a tiny gap called the synapse between them. The impulse cannot cross electrically, so transmission is chemical:

  1. The action potential reaches the axon terminal.
  2. Vesicles release neurotransmitter molecules into the synaptic gap.
  3. The neurotransmitter diffuses across and binds to receptors on the next neuron.
  4. If enough binds, a new action potential is triggered in that neuron.
  5. The neurotransmitter is then broken down or reabsorbed so the synapse can reset.

Synapses ensure signals travel in one direction only, because only the terminal end releases neurotransmitter.

Exam-style practice questions

Practice questions written in the style of SACE Board exam questions on this dot point, with worked answer explainers. The year tag is the paper they imitate, not the source.

2019 SACE Stage 21 marksOxycodone inhibits the release of neurotransmitters such as acetylcholine. Which one of the following is least likely to be affected by this drug: the transmission of a nerve impulse along the axon; the number of nerve impulses crossing the synapse; the level of pain suffered by a patient; or the number of membrane receptors that have acetylcholine bound to them?
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The least affected is the transmission of a nerve impulse along the axon. Conduction of the action potential along an axon is electrical and does not depend on neurotransmitter release, so blocking neurotransmitter release does not change it. By contrast, reducing acetylcholine release lowers the number of impulses crossing the synapse, reduces how many receptors have acetylcholine bound, and changes the pain signal reaching the brain.

2018 SACE Stage 21 marksCaffeine reduces the effect of the neurotransmitter adenosine (which induces sleep) by preventing adenosine from binding to receptors in the hypothalamus. Caffeine therefore: decreases the time to fall asleep; binds to the active site of adenosine; reduces the number of nerve impulses triggered by adenosine; or prevents the release of adenosine?
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Caffeine reduces the number of nerve impulses triggered by adenosine. By blocking adenosine receptors, caffeine stops adenosine from binding and producing its normal effect, so fewer of the impulses that adenosine would have triggered are generated. It does not bind adenosine's active site, does not block adenosine's release, and (being a stimulant) would increase, not decrease, the time taken to fall asleep.